Heat stress induces the richness and reproductive domesticated animal’s performance by settling the physiology conceptive steps, through hormonal irregularity, diminished oocyte quality and feeble semen quality, and diminished undeveloped organism advancement and endurance. It depends on principally milk production, nutrition, disease management, sexual activities, and heat stress tolerance capacity in livestock farming. The decreases infertility caused by elevated blood heat influences sex gland regulation, oestrus regulation, and gametocyte disturbance and also affects embryonic development. Heat stress reduces the degree of dominance of the seminal vesicles and this may be observed as reduced steroidogenic capability of its theca and granulose cells as fall in blood oestrogen concentrations. Plasma progestin levels are also diminished counting on whether or not the heat stress is acute and on the metabolic state of the animal. The endocrine changes the cyst activities and alters the ovulatory mechanism leading to a decrease in gametocyte and embryo quality. Summer infertility may be countered through oestrus behaviour can be mitigated by with the help of implementation of ovulation phase treatments to limited period of embryonic transfer and also advanced reproductive technologies involving hormonal treatments, systematic artificial insemination and which may enhance the possibility of establishing pregnancy in domestic animals.
Part of the book: Bovine Science